Data/hora: 08/06/2024 12:12:43

Provedor de dados: 	80	
País: 	Brazil	
Título: 	Envenoming by Viridovipera stejnegeri snake: a patient with liver cirrhosis presenting disruption of hemostatic balance	
Autores: 	Chien,Chih-Ying; Liao,Shu-Chen; Liao,Chien-Hung; Huang,Ting-Shuo; Chen,Yu-Hsien.	
Data: 	2017-01-01	
Ano: 	2017	
Palavras-chave: 	Snakebite; Liver cirrhosis; Coagulopathy.	
Resumo: 	Abstract  Background  In most cases of envenoming by the green habu Viridovipera stejnegeri in Taiwan coagulopathy is not observed.  Case presentation  Herein, we describe the case of a patient with liver cirrhosis who developed venom-induced consumptive coagulopathy after V. stejnegeri bite. Laboratory investigation revealed the following: prothrombin time &gt; 100 s (international normalized ratio &gt; 10), activated partial thromboplastin time &gt; 100 s, fibrinogen &lt; 50 mg/dL, and fibrin degradation product &gt; 80 μg/mL. The patient recovered after administration of bivalent hemorrhagic antivenom, vitamin K, fresh frozen plasma and cryoprecipitate.  Conclusion  The liver, directly involved in the acute phase reaction, is the main responsible for neutralization of animal toxins. Any patient with history of liver cirrhosis bitten by a venomous snake, even those whose venoms present low risk of coagulopathy, should be very carefully monitored for venom-induced consumptive coagulopathy (VICC), since the hemostatic balance may be disrupted.	
Tipo: 	Info:eu-repo/semantics/report	
Idioma: 	Inglês	
Identificador: 	http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1678-91992017000100501	
Editor: 	Centro de Estudos de Venenos e Animais Peçonhentos	
Relação: 	10.1186/s40409-017-0096-9	
Formato: 	text/html	
Fonte: 	Journal of Venomous Animals and Toxins including Tropical Diseases  v.23 2017	
Fonte: 	info:eu-repo/semantics/openAccess