Data/hora: 27/07/2024 02:01:01

Provedor de dados: 	87	
País: 	Brazil	
Título: 	A Modified phosphate-carrier protein theory is proposed as a non-target site mechanism For glyphosate resistance in weeds	
Autores: 	Roso,A.C.; Vidal,R.A.	
Data: 	2010-01-01	
Ano: 	2010	
Palavras-chave: 	Membrane carrier proteins; Herbicide; Weed resistance.	
Resumo: 	Glyphosate is an herbicide that inhibits the enzyme 5-enolpyruvyl-shikimate-3-phosphate synthase (EPSPs) (EC 2.5.1.19). EPSPs is the sixth enzyme of the shikimate pathway, by which plants synthesize the aromatic amino acids phenylalanine, tyrosine, and tryptophan and many compounds used in secondary metabolism pathways. About fifteen years ago it was hypothesized that it was unlikely weeds would evolve resistance to this herbicide because of the limited degree of glyphosate metabolism observed in plants, the low resistance level attained to EPSPs gene overexpression, and because of the lower fitness in plants with an altered EPSPs enzyme. However, today 20 weed species have been described with glyphosate resistant biotypes that are found in all five continents of the world and exploit several different resistant mechanisms. The survival and adaptation of these glyphosate resistant weeds are related toresistance mechanisms that occur in plants selected through the intense selection pressure from repeated and exclusive use of glyphosate as the only control measure. In this paper the physiological, biochemical, and genetic basis of glyphosate resistance mechanisms in weed species are reviewed and a novel and innovative theory that integrates all the mechanisms of non-target site glyphosate resistance in plants is presented.	
Tipo: 	Info:eu-repo/semantics/article	
Idioma: 	Inglês	
Identificador: 	http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-83582010000500025	
Editor: 	Sociedade Brasileira da Ciência das Plantas Daninhas	
Relação: 	10.1590/S0100-83582010000500025	
Formato: 	text/html	
Fonte: 	Planta Daninha  v.28 n.spe 2010	
Fonte: 	info:eu-repo/semantics/openAccess