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Registros recuperados: 4
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Cellular and humoral local immune responses in sheep experimentally infected with Oestrus ovis (Diptera: Oestridae) Inra
Tabouret, G.; Lacroux, C.; Andréoletti, O.; Bergeaud, J. P.; Hailu-Tolosa, Y.; Hoste, H.; Prévot, F.; Grisez, C.; Dorchies, P.; Jacquiet, P..
Tipo: Journal Article Palavras-chave: OESTRUS OVIS; REPONSE IMMUNITAIRE; LYMPHOCYTE; EOSINOPHILE; IMMUNOGLOBULINE A; IMMUNOGLOBULINE B; OESTRIDAE; DIPTERA; INFECTION; PATHOLOGIE INFECTIEUSE; LEUCOCYTE; GRANULOCYTE; CAVITE NASALE; ANTIGENE ANTICORPS; METHODE STATISTIQUE.
Ano: 2003 URL: http://www.prodinra.inra.fr/prodinra/pinra/doc.xsp?id=PUB0400023306102147&uri=/notices/prodinra1/2009/12/
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Functional genomic studies of the intestinal response to a foodborne enteropathogen in a humanized gnotobiotic mouse model Inra
Lecuit, M.; Sonnenburg, J. L.; Cossart, P.; Gordon, J. I..
Members of the genus Listeria provide a model for defining host responses to invasive foodborne enteropathogens. Active translocation of Listeria monocytogenes across the gut epithelial barrier is mediated by interaction of bacterial internalin (InlA) and its species-specific host receptor, E-cadherin, whereas translocation across Peyer’s patches through M-cells is InlA independent. To define microbial determinants and molecular correlates of the host response to translocation via these two routes, we colonized germ-free transgenic mice expressing the human enterocyte-associated E-cadherin receptor with wildtype (WT) or mutant L. monocytogenes strains, or its nonpathogenic noninvasive relative Listeria innocua, or with Bacteroides thetaiotaomicron, a...
Tipo: Journal Article Palavras-chave: LISTERIA MONOCYTOGENES; CADHERINE; BARRIERE INTESTINALE; LAMINA PROPRIA; PROTEINE BACTERIENNE; INTESTIN; MODELE ANIMAL; SOURIS; INFECTION; ENTEROPATHOGENE; MUTATION; EXPRESSION DES GENES; REPONSE IMMUNITAIRE; IMMUNITE.
Ano: 2007 URL: http://www.prodinra.inra.fr/prodinra/pinra/doc.xsp?id=PROD200924d16b14&uri=/notices/prodinra1/2010/11/
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Gene expression profiling of Spodoptera frugiperda hemocytes and fat body using cDNA microarray reveals polydnavirus-associated variations in lepidopteran host genes transcript levels Inra
Barat-Houari, M.; Hilliou, F.; Jousset, O.X.; Sofer, L.; Deleury, E.; Rocher, J.; Ravallec, M.; Galibert, L.; Delobel, P.; Feyereisen, R.; Fournier, P.; Volkoff, A.N..
Tipo: Journal Article Palavras-chave: RELATION HOTE PARASITOIDE; REPONSE IMMUNITAIRE; EXPRESSION GENIQUE SPODOPTERA FRUGIPERDA; LEPIDOPTERA.
Ano: 2006 URL: http://www.prodinra.inra.fr/prodinra/pinra/doc.xsp?id=PROD200716dc6408&uri=/notices/prodinra1/2011/06/
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Spermine causes loss of innate immune response to helicobacter pylori by inhibition of inducible nitric-oxide synthase translation Inra
Bussière, F.I.; Chaturvedi, R.; Cheng, Y.; Gobert, A.; Asim, M.; Blumberg, D.R.; Xu, H.; Kim, P.Y.; Hacker, A.; Casero, R.A.; Wilson, K.T..
Helicobacter pylori infection of the stomach elicits a vigorous but ineffective host immune and inflammatory response, resulting in persistence of the bacterium for the life of the host. We have reported that in macrophages, H. pylori up-regulates inducible NO synthase (iNOS) and antimicrobial NO production, but in parallel there is induction of arginase 11, generating ornithine, and of ornithine decarboxylase (ODC), generating polyamines. Spermine, in particular, has been shown to restrain immune response in activated macrophages by inhibiting proinflammatory gene expression. We hypothesized that spermine could prevent the antimicrobial effects of NO by inhibiting iNOS in macrophages activated by H. pylori. Spermine did not affect the upregulation of iNOS...
Tipo: Journal Article Palavras-chave: SPERMINE; REPONSE IMMUNITAIRE; OXYDE NITRIQUE; ORNITHINE DECARBOXYLASE; ESTOMAC; INFECTION; ARGINASE HELICOBACTER PYLORI.
Ano: 2005 URL: http://www.prodinra.inra.fr/prodinra/pinra/doc.xsp?id=PROD20089e9b2f6c&uri=/notices/prodinra1/2010/11/
Registros recuperados: 4
Primeira ... 1 ... Última
 

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