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Provedor de dados:  Genet. Mol. Biol.
País:  Brazil
Título:  Genes associated with hypersensitive response (HR) in the citrus EST database (CitEST)
Autores:  Guidetti-Gonzalez,Simone
Freitas-Astúa,Juliana
Amaral,Alexandre Morais do
Martins,Natália F.
Mehta,Angela
Silva,Marilia Santos
Carrer,Helaine
Data:  2007-01-01
Ano:  2007
Palavras-chave:  Otic and abiotic stress response
Plant-pathogen incompatible interaction
Programmed cell death
Plant disease resistance
Resumo:  Plants are continuously exposed to pathogen attack, but successful infection is rare because they protect themselves against pathogens using a wide range of response mechanisms. One of them is the hypersensitive response (HR), which is a form of cell death often associated with plant resistance to pathogen infection to prevent the spreadsebpg@cnpq.br sebpg@cnpq.br of the potential pathogen from infected to uninfected tissues. Cell death is activated by recognition of pathogen-derived molecules by the resistance (R) gene products, and is associated with the massive accumulation of reactive oxygen species (ROS), salicylic acid (SA), and other pro-death signals such as nitric oxide (NO). The analysis of the citrus EST (CitEST) database revealed the presence of putative genes likely to be involved in HR through their products, like metacaspases, lipoxygenases, phospholipases, pathogenesis-related proteins, glutathione transferases/peroxidases, enzymes involved in the phenylpropanoid pathway and in the formation and detoxification of ROS, as well as those involved in the formation and regulation of ion channels, SA and NO. By analysis of the EST database of Citrus, it was possible to identify several putative genes that code for key enzymes involved in HR triggering and also in plant defense against biotic and abiotic stress.
Tipo:  Info:eu-repo/semantics/other
Idioma:  Inglês
Identificador:  http://www.scielo.br/scielo.php?script=sci_arttext&pid=S1415-47572007000500022
Editor:  Sociedade Brasileira de Genética
Relação:  10.1590/S1415-47572007000500022
Formato:  text/html
Fonte:  Genetics and Molecular Biology v.30 n.3 suppl.0 2007
Direitos:  info:eu-repo/semantics/openAccess
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