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	Provedor de dados BJMBR (3) Autor Chagas,C.A. (1) Godoy Jr.,C.E. (1) Godoy,C.E. (1) Ladeira,M.S.P. (1) Magalhães,A. (1) Metze,K. (1) Pedrazzoli Jr,J. (1) Reis,C.A. (1) Ribeiro,M.L. (1) Santos,J.C. (1) Trevisan,M.A.S. (1) Vassallo,J. (1) Mais... Palavra-chave Chronic gastritis (3) Helicobacter pylori (2) Apoptosis (1) Bacterial adhesins (1) CD95 (1) FAS/APO1 (1) Gastric cancer (1) Gastric carcinogenesi (1) Glycan receptors (1) IL-1RN (1) IL-1β (1) Interleukins (1) Lewis antigens (1) MALT lymphoma (1) Polymorphisms (1) TNF-α (1) Mais... Tipo do documento Info:eu-repo/semantics/article (3) Ano 2012 (1) 2010 (1) 2004 (1) País Brazil (3) Idioma Inglês (3)		Ordenar por:  Relevância Autor Título Ano Registros recuperados: 3 Primeira ... 1 ... Última Helicobacter pylori adhesion to gastric epithelial cells is mediated by glycan receptors BJMBR Magalhães,A.; Reis,C.A.. Helicobacter pylori adhesion to gastric epithelial cells constitutes a key step in the establishment of a successful infection of the gastric mucosa. The high representation of outer membrane proteins in the bacterial genome suggests the relevance of those proteins in the establishment of profitable interactions with the host gastric cells. Gastric epithelial cells are protected by a mucous layer gel, mainly consisting of the MUC5AC and MUC6 mucins. In addition to this protective role, mucins harbor glycan-rich domains that constitute preferential binding sites of many pathogens. In this article we review the main players in the process of H. pylori adhesion to gastric epithelial cells, which contribute decisively to the high prevalence and chronicity of... Tipo: Info:eu-repo/semantics/article Palavras-chave: Helicobacter pylori; Bacterial adhesins; Lewis antigens; Glycan receptors; Chronic gastritis; Gastric carcinogenesi. Ano: 2010 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2010000700002 Immunoexpression of CD95 in chronic gastritis and gastric mucosa-associated lymphomas BJMBR Vassallo,J.; Godoy Jr.,C.E.; Godoy,C.E.; Chagas,C.A.; Metze,K.; Trevisan,M.A.S.. CD95 (Fas/APO-1)-mediated apoptosis plays an important role in immunological regulation and is related to the pathogenesis of autoimmune diseases. Immunoexpression of CD95 has been reported to frequently occur in low grade non-Hodgkin lymphomas, especially of post-germinal center histogenesis, among which those originating in mucosa-associated lymphoid tissue (MALT lymphomas). However, there is no report comparing in situ immunoexpression of this marker in lymphomas and the hyperplastic lymphoid reaction (chronic gastritis) related to Helicobacter pylori infection. The purpose of the present research was to compare the intensity of lymphoid CD95 immunoexpression in 15 cases of H. pylori-related chronic gastritis and 15 gastric MALT lymphomas. CD95... Tipo: Info:eu-repo/semantics/article Palavras-chave: CD95; FAS/APO1; Apoptosis; Chronic gastritis; MALT lymphoma. Ano: 2004 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2004000900015 Relationship of IL-1 and TNF-α polymorphisms with Helicobacter pylori in gastric diseases in a Brazilian population BJMBR Santos,J.C.; Ladeira,M.S.P.; Pedrazzoli Jr,J.; Ribeiro,M.L.. It is well known that the risk of development of gastric cancer (GC) in Helicobacter pylori-infected patients depends on several factors. Thus, the aim of this study was to investigate the effect of proinflammatory cytokine gene polymorphisms for IL-1β, IL-1RN and TNF-α on the development of GC in a Brazilian population. A total of 202 biopsies obtained from Brazilian patients with chronic gastritis and GC were included in the study. Infection with H. pylori cagA+ was determined by the polymerase chain reaction (PCR) as previously described. IL-1β, IL-1RN and TNF-α polymorphism genotyping was performed by restriction fragment length polymorphism PCR. Associations between gene polymorphisms, clinical diseases and virulence markers were evaluated using... Tipo: Info:eu-repo/semantics/article Palavras-chave: Chronic gastritis; Gastric cancer; Polymorphisms; Helicobacter pylori; Interleukins; IL-1β; IL-1RN; TNF-α. Ano: 2012 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2012000900004 Registros recuperados: 3 Primeira ... 1 ... Última

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