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	Provedor de dados BJMBR (3) Autor Liu,J. (3) Chen,J. (1) Gao,T.X. (1) Gao,Y.C. (1) Guo,B.Y. (1) He,J. (1) Li,L. (1) Li,X.H. (1) Li,Z. (1) Liu,G.J. (1) Liu,Z.H. (1) Ma,F.Y. (1) Meng,Q. (1) Nikolic-Paterson,D.J. (1) Ran,J.H. (1) Wang,B. (1) Zhai,S. (1) Zhang,X.L. (1) Zhu,Y. (1) Mais... Palavra-chave Aortic dissection (1) Apoptosis (1) C-Jun amino terminal kinase (1) Differentiation (1) Endovascular aortic repair (1) Fibrosis (1) Hepatic oval cells (1) Inflammation (1) Liver transplantation (1) Open surgical repair (1) P38 (1) Proliferation (1) Renal pathophysiology (1) Type-B aortic dissection (1) Mais... Tipo do documento Info:eu-repo/semantics/article (3) Ano 2016 (1) 2013 (1) 2009 (1) País Brazil (3) Idioma Inglês (3)		Ordenar por:  Relevância Autor Título Ano Registros recuperados: 3 Primeira ... 1 ... Última Long-term efficacy of endovascular vs open surgical repair for complicated type-B aortic dissection: a single-center retrospective study and meta-analysis BJMBR Zhu,Y.; Wang,B.; Meng,Q.; Liu,J.; Zhai,S.; He,J.. This study aimed to evaluate the long-term survival and risk factors of traditional open surgical repair (OSR) vs thoracic endovascular aneurysm repair (TEVAR) for complicated type-B aortic dissection (TBAD). A total of 118 inpatients (45 OSR vs 73 TEVAR) with TBAD were enrolled from January 2004 to January 2015. Kaplan-Meier curves and Cox proportional hazards analysis were performed to identify the long-term survival rate and independent predictors of survival, respectively. Meta-analysis was used to further explore the long-term efficacy of OSR and TEVAR in the eight included studies using Review Manager 5.2 software. An overall 10-year survival rate of 41.9% was found, and it was similar in the two groups (56.7% OSR vs 26.1% TEVAR; log-rank P=0.953).... Tipo: Info:eu-repo/semantics/article Palavras-chave: Aortic dissection; Open surgical repair; Type-B aortic dissection; Endovascular aortic repair. Ano: 2016 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2016000600707 In vitro proliferation and differentiation of hepatic oval cells and their potential capacity for intrahepatic transplantation BJMBR Li,Z.; Chen,J.; Li,L.; Ran,J.H.; Liu,J.; Gao,T.X.; Guo,B.Y.; Li,X.H.; Liu,Z.H.; Liu,G.J.; Gao,Y.C.; Zhang,X.L.. Hepatic oval cells (HOCs) are recognized as facultative liver progenitor cells that play a role in liver regeneration after acute liver injury. Here, we investigated the in vitro proliferation and differentiation characteristics of HOCs in order to explore their potential capacity for intrahepatic transplantation. Clusters or scattered HOCs were detected in the portal area and interlobular bile duct in the liver of rats subjected to the modified 2-acetylaminofluorene and partial hepatectomy method. Isolated HOCs were positive for c-kit and CD90 staining (99.8% and 88.8%, respectively), and negative for CD34 staining (3.6%) as shown by immunostaining and flow cytometric analysis. In addition, HOCs could be differentiated into hepatocytes and bile duct... Tipo: Info:eu-repo/semantics/article Palavras-chave: Hepatic oval cells; Proliferation; Differentiation; Liver transplantation. Ano: 2013 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2013000800681 The role of stress-activated protein kinase signaling in renal pathophysiology BJMBR Ma,F.Y.; Liu,J.; Nikolic-Paterson,D.J.. Two major stress-activated protein kinases are the p38 mitogen-activated protein kinase (MAPK) and the c-Jun amino terminal kinase (JNK). p38 and JNK are widely expressed in different cell types in various tissues and can be activated by a diverse range of stimuli. Signaling through p38 and JNK is critical for embryonic development. In adult kidney, p38 and JNK signaling is evident in a restricted pattern suggesting a normal physiological role. Marked activation of both p38 and JNK pathways occurs in human renal disease, including glomerulonephritis, diabetic nephropathy and acute renal failure. Administration of small molecule inhibitors of p38 and JNK has been shown to provide protection from renal injury in different types of experimental kidney disease... Tipo: Info:eu-repo/semantics/article Palavras-chave: P38; C-Jun amino terminal kinase; Inflammation; Fibrosis; Apoptosis; Renal pathophysiology. Ano: 2009 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2009000100006 Registros recuperados: 3 Primeira ... 1 ... Última

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