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	Provedor de dados Acta Toxicol. Argent. (1) BJMBR (1) Autor Ayinbuomwan,Stephen A (1) Chedid,N.G.B. (1) Díaz G.,L.E. (1) Isah,Ambrose Ohumagho (1) Masuda,M.O. (1) Oghuvwu,Sunday Omokiniovo (1) Opadeyi,Abimbola (1) Quintero H.,C.C. (1) Saraiva,R.M. (1) Mais... Palavra-chave Infarction (2) Acute (1) Adrenergic receptor (1) Ca2+ channel (1) Carbon monoxide (1) Heart failure (1) Hypertrophy (1) Poisoning (1) Mais... Tipo do documento Info:eu-repo/semantics/article (1) Info:eu-repo/semantics/report (1) Ano 2018 (1) 2003 (1) País Argentina (1) Brazil (1) Idioma Inglês (2)		Ordenar por:  Relevância Autor Título Ano Registros recuperados: 2 Primeira ... 1 ... Última Acute carbon monoxide poisoning with coexisting cerebral and myocardial infarction: The brain-heart connection and challenges of management in resource-limited settings Acta Toxicol. Argent. Oghuvwu,Sunday Omokiniovo; Isah,Ambrose Ohumagho; Opadeyi,Abimbola; Ayinbuomwan,Stephen A. Carbon monoxide poisoning pose significant public health consequence and management can be challenging in resource-poor countries. This is a commentary of a 40 year-old farmer with a five day history of loss of consciousness following overnight inhalation of carbon monoxide. Neuroimaging and electrophysiologic studies showed bilateral internal capsule and myocardial infarction. He had unsuccessful treatment with normobaric oxygen therapy in the absence of hyperbaric oxygen. This case depicts the brain-heart connection in carbon monoxide poisoning and highlights the challenges of management in a resource poor setting. Tipo: Info:eu-repo/semantics/report Palavras-chave: Carbon monoxide; Poisoning; Acute; Infarction. Ano: 2018 URL: http://www.scielo.org.ar/scielo.php?script=sci_arttext&pid=S1851-37432018000200004 Impaired beta-adrenergic response and decreased L-type calcium current of hypertrophied left ventricular myocytes in postinfarction heart failure BJMBR Saraiva,R.M.; Chedid,N.G.B.; Quintero H.,C.C.; Díaz G.,L.E.; Masuda,M.O.. Infarct-induced heart failure is usually associated with cardiac hypertrophy and decreased ß-adrenergic responsiveness. However, conflicting results have been reported concerning the density of L-type calcium current (I Ca(L)), and the mechanisms underlying the decreased ß-adrenergic inotropic response. We determined I Ca(L) density, cytoplasmic calcium ([Ca2+]i) transients, and the effects of ß-adrenergic stimulation (isoproterenol) in a model of postinfarction heart failure in rats. Left ventricular myocytes were obtained by enzymatic digestion 8-10 weeks after infarction. Electrophysiological recordings were obtained using the patch-clamp technique. [Ca2+]i transients were investigated via fura-2 fluorescence. ß-Adrenergic receptor density was... Tipo: Info:eu-repo/semantics/article Palavras-chave: Ca2+ channel; Heart failure; Hypertrophy; Infarction; Adrenergic receptor. Ano: 2003 URL: http://www.scielo.br/scielo.php?script=sci_arttext&pid=S0100-879X2003000500012 Registros recuperados: 2 Primeira ... 1 ... Última

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