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Interleukin-6 activates arginine vasopressin neurons in the supraoptic nucleus during immune challenge in rats Inra
Palin, K.; Moreau, M. L..; Sauvant, J.; Orcel, H.; Nadjar, A.; Duvoid-Guillou, A.; Dudit, J.; Rabié, A.; Moos, F..
The increase of plasma arginin-vasopressin (AVP) release, which translates hypothalamic AVP neuron activation in response to immune challenge, appears to occur independently of plasma osmolality or blood pressure changes. Many studies have shown that major inflammatory mediators produced in response to peripheral inflammation, such as prostaglandin (PG)-E2 and interleukin (IL)-1β, excite AVP neurons. However, in vivo electrical activation of AVP neurons was still not assessed in relation to plasma AVP release, osmolality, or blood pressure or to the expression and role of inflammatory molecules like PG-E2, IL-1β, IL-6, and tumor necrosis factor-{alpha} (TNF{alpha}). This study aims at elucidating those factors that underlie the activation of AVP neurons in...
Tipo: Journal Article Palavras-chave: HORMONE ANTIDIURÉTIQUE; NEURONE; CERVEAU; DIURÈSE; OSMOLALITÉ; PRESSION ARTÉRIELLE; NOYAU SUPRA-OPTIQUE; PLASMA; PROSTAGLANDINE; INTERLEUKINE; FACTEUR DE NÉCROSE TUMORALE; ACTIVATION DES NEURONES LIPOPOLYSACCHARIDE INDUCED-HYPOTHALAMIC VASOPRESSIN NEURON ACTIVATION; BRAIN CYTOKINES; DIURESIS; PLASMA OSMOLALITY; BLOOD PRESSURE.
Ano: 2009 URL: http://www.prodinra.inra.fr/prodinra/pinra/doc.xsp?id=PROD2009179193ce&uri=/notices/prodinra1/2010/09/
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